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Lipoid Nephrosis

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1- Biology:

A- Definition:

Massive proteinuria> 50 mg / kg / day (40 mg / m² / h); clear

Hypoproteinemia and hypoalbuminemia (<30 g / L)

Hyperlipidemia and hypercholesterolemia

B- Protein:

α2-globulins still high (-> 30% plasma protein)

Gamma globulins are low

High coagulation factors

Reduce protein vector => anemia (decreased transferrin) hypothyroidism

C- Electrolytes:

Natriuresis is always reduced (aldosteronism 2aire)

Normal or low serum sodium; normal or increased serum potassium

Decreased total serum calcium (decreased albumin).

D- Blood:

Hypovolemia

Thrombocytosis and anemia (discrete); Leukocytosis

Elevated ESR

Hyperlipidemia is due to the decrease of orosomucoid (factor of lipoprotein lipase)

Lipoid nephrosis -> 3 microscopic type:

Minimal glomerular lesions

Injury focal segmental glomerulosclerosis

Mesangial proliferation

Urinary symptoms: frequent microscopic hematuria; oliguria

Functional renal impairment is rare, associated with significant oliguria

The decrease in gamma globulin covers IgG whereas IgM are increased

C3 fraction of complement is normal

Lipoid nephrosis

2- Complications:

A- Infectious:

Pneumococcal disease (pneumonia, meningitis, peritonitis)

Skin infections (pyoderma …) …

B- Hydro-sodées:

Fluid retention: hyponatremia, headache and convulsion

By sudden collapse hypovolemia (beware diuretics)

C- Thrombo-embolism:

Renal vein thrombosis …

Pulmonary embolism

D- Digestive:

Diarrhea

Abdominal pain: serous edema, ascites

E- Other:

Malnutrition; hypothyroidism

Convulsion (hyponatremia, hypocalcemia); tetany

Congenital nephrotic syndrome of the Finnish-type (before 1 year, severe)

Secondary SN: Berger’s disease (IgA deposition); HSP; hepatitis B …

Blood pressure is usually normal

Symptomatic treatment: without strict salt diet edema phase; normal drink (water restriction and diuretics should be avoided)

For large edema => albumin + furosemide

Total length of treatment with prednisone => 4 months and ½

The disappearance of the clinical SN and proteinuria after 1 continuous attack treatment months defines the cortico-sensitivity

Corticosteroid: relapse after stopping corticosteroids or when decreasing doses

Healing: absence of clinical and biological signs for 2 years without corticosteroids.

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